To understand the pathogenesis of desquamative gingivitis, first, we must know the basic organization of the oral mucosa. OLP is usually asymptomatic, although when there are areas of erosion or ulceration, the patient may have variable amounts of discomfort, being particularly troublesome when eating spicy or acidic type foods. Some cases show fibrinogen and fibrin deposition in a linear pattern in the basement membrane zone. Painful, solitary small blisters and erosions with surrounding erythema are present mainly on the gingiva and the lateral border of the tongue. In patients with HSV infection associated recurrent EM and idiopathic recurrent EM, the first-line treatment is antiviral prophylaxis , , These include aromatic compounds found in chewing gums and toothpastes, most common being carvone, spearmint essential oil, menthol essential oil, cinnamaldehyde, and cinnamon essential oil Roche C, Field EA. 
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Bullous pemphigoid, mucous membrane pemphigoid and pemphigoid. The histopathological dessuamative shows intraepithelial blister formation associated with extensive acantholysis of keratinocytes 45, Nisengard RJ, Neiders M. Dapsone or sulfapyridine are the treatments of choice for non drug-related dermatitis. After initiation of corticosteroid therapy, clinical improvement may be seen within a few weeks. Desquamative gingivitis is a complex term that needs a clear definition.
DG can be caused by numerous conditions. The mainstream therapy of these gingival conditions is still local and systemic corticosteroid therapy.
EM is an acute mucocutaneous disorder, characterized by varying degrees of blistering and ulceration. Non-specific mechanisms in oral lichen dssquamative The intraepithelial vesiculation begins as a microscopic alteration.
Desquamative gingivitis
Linear IgA bullous disease affects adults of all ages, but is most commonly seen in the patients older than 30 years Hemidesmosomes are involved in cell and basement membrane contact, linking the keratinocyte cytoskeletons to the lamina lucida. Presently, many systemic autoimmune disorders and other conditions have been described to have desquamative gingival lesions. Increased concentrations of MMP-9 have been found to be increased in culture supernatants taken from patients with OLP as compared to normal controls Histopathological findings include hyper-orthokeratosis, acanthosis or atrophy and liquefaction degeneration of the basal layer of the affected epithelium.
It is likely desquamayive BP is a result of the breakdown of peripheral tolerance to BP antigens.
Desquamative Gingivitis
Some of these can be used as diagnostic markers, while others can help in quantifying the disease activity. Adjuvant drugs having steroid sparing action are also commonly used desqjamative the aim of increasing efficacy of the treatment. Temporomandibular jointsmuscles of mastication and malocclusions — Jaw joints, chewing muscles and bite abnormalities.
This involves checking out of the following points: How to do research? To a lesser extent C3 can also be found deposited linearly at the basement membrane zone. Aging Aging results in thinning of the oral epithelium and diminished keratinization.
Desquamative Gingivitis - ppt download
Cementicle Cementoblastoma Gigantiform Cementoma Eruption cyst Epulis Pyogenic granuloma Congenital epulis Gingival enlargement Gingival cyst of the adult Gingival cyst of the newborn Gingivitis Desquamative Granulomatous Plasma cell Hereditary gingival fibromatosis Hypercementosis Hypocementosis Linear gingival erythema Necrotizing periodontal diseases Acute necrotizing ulcerative gingivitis Pericoronitis Peri-implantitis Periodontal abscess Periodontal trauma Periodontitis Aggressive As a manifestation of systemic disease Chronic Perio-endo lesion Teething.
Oral lesions mostly manifest as scattered painful erosions or desquamative gingivitis with smooth erosions along the attached gingiva or hingivitis mucosal detachment. Mast cells are increased in OLP lesion.
Successful treatment of the oral lesions in mucous membrane pemphigoid has been reported with the use of topical steroids, tacrolimus, and cyclosporine Its nature and treatment. Conventional therapy Debridement Scaling and root planing Full mouth disinfection Full mouth ultrasonic debridement.
The oral lesions of pemphigus vulgaris may also be in the form of flat erosions, which may be easily misdiagnosed as denture sores in the denture wearing patients.
In certain patients only oral lesions may be present. There is no specific treatment for erythema multiforme. The adult dose of dapsone is mg per day with reduced dose in children.
Analgesics may be given to control pain. The etiopathogenesis of OLP has been a matter of controversy.
Six students of this group were randomly assigned to use the flowchart.
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